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ABSTRACT
Preterm birth (PTB) is the world’s leading cause of neonatal morbidity and mortality, accounting for 35% of annual neonatal deaths. Uterine infection and inflammation are associated with 40% of PTB cases. Currently, available therapies are mainly tocolytics which have been largely ineffective with accompanying adverse outcomes. Chlorophyll derivatives exert uterine-modulating effects, including anti-inflammatory and antioxidant activities. Therefore, the study aimed at investigating the effects of pheophorbide a (PE) and pheophytin a (PT) on lipopolysaccharide (LPS)-induced preterm labour in mouse models.
